Which statement is correct

Correct Answer: A

Rationale: Glucagon raises glucose via glycogenolysis without increasing lactate gluconeogenesis (lactate source) is secondary. Insulin uses GLUT2 in liver, not GLUT4 (muscle/adipose). Cortisol boosts gluconeogenesis, not glycogenolysis primarily. Glucagon stimulates insulin short-term, not inhibits. Glucagon's lactate-free glucose rise distinguishes it, key to its hepatic action, unlike transporter, cortisol, or insulin errors.

Correct Answer: D

Rationale: T4 increases cardiovascular β-receptors, enhancing catecholamine sensitivity metabolic/cardiac boost. It's regulated by negative feedback (TSH, TRH). Most T4 binds TBG (~70%), not albumin (~10%). T4 lowers LDL by increasing clearance, not raising it. β-receptor increase distinguishes T4's action, critical for heart response, unlike feedback, transport, or lipid errors.

Correct Answer: D

Rationale: Hormones regulate internal environment (e.g., ADH for water), metabolism (e.g., thyroxine), glandular secretions (e.g., TSH), and growth (e.g., GH), but they don't produce electrolytes ions like sodium or potassium are managed by kidneys or diet, not synthesized by hormones. 'Produces electrolytes' is incorrect as hormones modulate existing substances, not create them. This distinction highlights hormones' role as signaling molecules, not producers of biochemical raw materials, critical for understanding endocrine specificity versus metabolic processes.

Correct Answer: A

Rationale: Luteinizing hormone (LH) from the anterior pituitary triggers gonadal secretion of progesterone (ovaries) and testosterone (testes), not TSH (thyroid), corticotropin (adrenals), MSH, or GH. LH's reproductive role distinguishes it, essential for sex hormone cycles, contrasting with metabolic or pigment regulators.

Correct Answer: C

Rationale: Aldosterone hyposecretion reduces sodium retention, lowering blood volume and pressure, leading to dehydration not heart rate, airway, oxygen, or pH changes directly. This distinguishes aldosterone's role in electrolyte balance, critical for hydration, contrasting with cardiovascular or acid-base regulators.