Which of the following is NOT true of growth hormone?

Correct Answer: C

Rationale: Growth hormone (GH) doesn't promote glucose metabolism it inhibits uptake, raising blood glucose (insulin antagonist), favoring fat metabolism (lipolysis) instead. It boosts amino acid entry and protein synthesis (growth). Glucose metabolism is insulin's domain GH spares it. Fat metabolism promotion distinguishes GH's role, key to its anabolic shift, unlike amino acid, protein, or glucose claims.

Correct Answer: D

Rationale: Aldosterone increases blood pressure by targeting kidney tubules for sodium/water reabsorption (less urine) and potassium excretion mineralocorticoid action. Decreasing blood pressure contradicts raises it via volume. Mineralocorticoid status and urine reduction are true. Pressure increase distinguishes aldosterone, critical for RAAS, unlike erroneous decrease.

Correct Answer: D

Rationale: Glucagon (pancreas) raises blood glucose via glycogenolysis and promotes fat/protein use (sparing glucose), but doesn't store glucose as glycogen insulin does that (glycogenesis). Storage contradicts glucagon's role mobilizes energy. Pancreatic origin, glucose increase, and sparing are true. Non-storage distinguishes glucagon, key to its catabolic action, unlike anabolic error.

Correct Answer: B

Rationale: Aldosterone (adrenal) regulates sodium/potassium, not fuel (glucose/fat) electrolyte focus. Insulin lowers glucose, glucagon raises it, cortisol mobilizes fuel (gluconeogenesis, lipolysis) all metabolic. Aldosterone's non-metabolic role distinguishes it, key to fluid balance, unlike glucose/fat-regulating hormones.

Correct Answer: D

Rationale: Pancreatic exocrine cells (acinar) produce digestive juices (e.g., amylase, lipase) via ducts aid digestion. Adrenaline (adrenal medulla) is endocrine, stress-related. Insulin (pancreatic β-cells) is endocrine, glucose-lowering, not exocrine. Cortisol (adrenal cortex) is steroid, not pancreatic. Digestive juices distinguish exocrine pancreas, key to nutrient breakdown, unlike endocrine stress, glucose, or steroid outputs.