Which of the following is NOT true of Cushing syndrome?

Correct Answer: D

Rationale: Cushing syndrome, from cortisol excess (adrenal cortex hypersecretion), causes trunk obesity, moon face, and buffalo hump not hyposecretion, which is Addison's. Basic blood pH (alkalosis) can occur from mineralocorticoid effects, true in severe cases. Hyposecretion contradicts excess defines it. Hypersecretion basis distinguishes Cushing's, key to its phenotype, unlike pH or hypo-error.

Correct Answer: D

Rationale: Steroid hormones, derived from cholesterol, include corticosteroids (e.g., cortisol), sex hormones (e.g., testosterone), and vitamin D lipid-based, nuclear-acting. GnRH, GHRH, TRH are peptides (hypothalamus), not steroids. GH and ACTH are pituitary peptides, not cholesterol-derived. T4/T3 are amines from tyrosine, not steroids. Corticosteroids, sex hormones, and vitamin D's cholesterol origin distinguishes them, key to their synthesis and action, unlike peptide or amine families.

Correct Answer: C

Rationale: Insulin lowers blood glucose by promoting glycogenesis (glucose to glycogen) and lipogenesis (fat synthesis) while inhibiting glycogenolysis (glycogen breakdown). Gluconeogenesis (glucose from non-carbs) is not an insulin function it's suppressed by insulin and stimulated by glucagon. This distinction is key: insulin drives storage, not glucose production, critical for post-meal glucose clearance. Misidentifying gluconeogenesis as an insulin role confuses its anabolic nature with catabolic processes, vital for NEET-level understanding of metabolic regulation.

Correct Answer: C

Rationale: Lacrimal glands secrete tears via ducts (exocrine), not hormones unlike adrenal (cortisol), pituitary (GH), and thyroid (T3/T4), which are ductless endocrine glands. This distinguishes exocrine vs. endocrine classification, critical for NEET's glandular taxonomy, avoiding confusion with secretory functions.