Which of the following hormones is produced by adipose tissue?

Correct Answer: A

Rationale: Adipose tissue produces leptin for appetite control, not insulin (pancreas) or others (mislisted organs). 'E' fits intent. This distinguishes leptin's metabolic role, vital for energy balance, contrasting with pancreatic or gonadal hormones.

Correct Answer: C

Rationale: Endocrine signaling uses blood vessels to transport hormones (e.g., cortisol) to targets, enabling systemic effects. Neurons signal electrically/chemically, not via blood transport distinct system. Ducts are exocrine (e.g., sweat glands), not endocrine. Membranes facilitate receptor binding, not transport. Blood vessels' role distinguishes endocrine reach, critical for widespread regulation, unlike neural, exocrine, or cellular mechanisms.

Correct Answer: B

Rationale: Negative feedback controls most endocrine secretions (e.g., high cortisol suppresses ACTH), maintaining homeostasis excess inhibits production. Positive feedback (e.g., oxytocin in labor) is rare, amplifying. Neural feedback is specific (e.g., medulla), not broad. Hormonal is a type, not the system. Negative feedback distinguishes endocrine stability, key to regulation, unlike amplifying or partial mechanisms.

Correct Answer: B

Rationale: Hypothalamus produces ADH (water) and oxytocin (labor), stored/released by posterior pituitary neurohypophyseal pair. Aldosterone is adrenal, not hypothalamic. Prolactin is anterior pituitary, not posterior. Cortisol is adrenal cortex, unrelated. ADH-oxytocin duo distinguishes posterior pituitary function, key to hypothalamic delivery, unlike adrenal or anterior hormones.

Correct Answer: C

Rationale: GH overproduction in adults causes acromegaly bone thickening (e.g., hands, face) post-epiphyseal closure. Dwarfism is childhood GH lack. Gigantism is pre-closure excess height surge. Myxedema is hypothyroidism, unrelated. Acromegaly's adult-specific bone changes distinguish it, key to GH excess timing, unlike childhood or thyroid conditions.