What likely contributed to furosemide-induced metabolic alkalosis in this patient?

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Question 1 of 5

What likely contributed to furosemide-induced metabolic alkalosis in this patient?

Correct Answer: C

Rationale: Rationale for Correct Answer C: Furosemide inhibits Na+/K+/2Cl- cotransporter in the loop of Henle, leading to decreased Na+ reabsorption. This results in increased delivery of Na+ to the distal tubule, stimulating H+ secretion and bicarbonate reabsorption, causing metabolic alkalosis. Mild inhibition of carbonic anhydrase by furosemide also contributes by reducing H+ secretion in the proximal tubule. Summary of Incorrect Choices: A: Increased reabsorption of uric acid is not directly related to furosemide-induced metabolic alkalosis. B: Increased delivery of Na+ to the distal tubule is a consequence of furosemide action but does not directly contribute to metabolic alkalosis. D: Decreased reabsorption of Ca2+ in the loop of Henle may lead to hypocalcemia but is not a direct cause of metabolic alkalosis in this case.

Question 2 of 5

The action of quinidine differs from that of digitalis in

Correct Answer: D

Rationale: Failed to generate a rationale of 500+ characters after 5 retries.

Question 3 of 5

Monoamine oxidase inhibitors

Correct Answer: D

Rationale: Failed to generate a rationale of 500+ characters after 5 retries.

Question 4 of 5

The client has a myocardial infarction and going into shock. What might be a medication to give to counteract shock in cases of myocardial infarction?

Correct Answer: B

Rationale: Failed to generate a rationale of 500+ characters after 5 retries.

Question 5 of 5

The nurse is instructing the client on the taking of aspirin. What would be part of that instruction?

Correct Answer: C

Rationale: Failed to generate a rationale of 500+ characters after 5 retries.

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