What is the appropriate priority nursing intervention?

Correct Answer: C

Rationale: The correct answer is C. Low potassium levels can increase the risk of digoxin toxicity. Checking the digoxin level is important to assess the patient's risk. Stopping the digoxin abruptly (choice D) can lead to serious consequences. Doing nothing (choice A) is negligent. Ordering potassium (choice B) without assessing the digoxin level first can be harmful. Prioritizing patient safety, checking the digoxin level is essential in this scenario.

Correct Answer: C

Rationale: The correct answer is C: Sodium channel blocker. Class II anti-arrhythmic agents primarily target beta blockers. Sodium channel blockers are classified as Class I anti-arrhythmic agents. These drugs block sodium channels in cardiac cells, which helps in regulating the heart's rhythm. Calcium channel blockers and ACE inhibitors do not directly target the heart's electrical activity, making them incorrect choices. Beta blockers are Class II anti-arrhythmic drugs, but since this question is asking for a different class of drug, B is not the correct answer.

Correct Answer: B

Rationale: The correct answer is B. A blood pressure of 145/95 would trigger the release of renin from the kidneys when a client is being treated with an ACE inhibitor. This is because this blood pressure reading indicates inadequate blood flow to the kidneys, leading to the activation of the renin-angiotensin-aldosterone system (RAAS) to increase blood pressure by vasoconstriction and fluid retention. Choices A, C, and D do not directly relate to the release of renin and the RAAS system in the context of hypertension treatment.

Correct Answer: B

Rationale: The correct answer is B: Give Protamine. Protamine is the antidote for heparin toxicity as it reverses its anticoagulant effects by forming a stable complex with heparin. Vitamin K (A) is used to reverse the effects of warfarin, not heparin. Warfarin (C) is another anticoagulant and would not be effective in counteracting heparin toxicity. Platelets (D) are not used to counteract heparin toxicity but may be indicated if the client is experiencing thrombocytopenia due to heparin-induced thrombocytopenia.

Correct Answer: A

Rationale: The correct answer is A: Aspirin. Aspirin is a platelet inhibitor because it irreversibly inhibits cyclooxygenase, reducing the formation of thromboxane A2 which is crucial for platelet aggregation. Pradaxa (B) is a direct thrombin inhibitor, Heparin (C) is an anticoagulant that works by enhancing antithrombin activity, and Warfarin (D) inhibits the synthesis of vitamin K-dependent clotting factors. Therefore, Aspirin is the only drug among the choices that directly inhibits platelet function.