ATI RN
ATI Pharmacology Proctored Exam 2019 Quizlet Questions
Question 1 of 5
The patient asks the nurse why she needs to continue using table salt because her prescribed lithium (Eskalith) is a salt. What is the best response by the nurse?
Correct Answer: D
Rationale: Lithium competes with sodium-adequate salt and hydration (1-1.5 L water) prevent toxicity by aiding excretion, per pharmacokinetics. Sea salt isn't less needed-sodium's the key. Retention isn't direct-hydration matters more. Increasing salt for toxicity is reactive, not preventive. Water trumps salt, clarifying use.
Question 2 of 5
A 27-year-old man with HIV disease and hepatitis B is hospitalized for treatment of his hepatitis B. He has begun on intravenous treatment with interferon. After administration, he develops fever, chills, and myalgias. Physical examination reveals that the lungs are clear to auscultation bilaterally. What is the most likely explanation for this reaction?
Correct Answer: A
Rationale: Failed to generate a rationale of 500+ characters after 5 retries.
Question 3 of 5
A 22-year-old woman ingests an entire bottle of acetaminophen in an attempted suicide. She unexpectedly feels well; and when her boyfriend discovers what she has done, he takes her to the emergency department. Which of the following drugs should be given in the ER?
Correct Answer: B
Rationale: Failed to generate a rationale of 500+ characters after 5 retries.
Question 4 of 5
Modest improvement in the memory of patients with Alzheimer's disease may occur with drugs that increase transmission at which of the following receptors?
Correct Answer: B
Rationale: Alzheimer's disease features cholinergic neuron loss, impairing memory via reduced acetylcholine signaling in the hippocampus and cortex. Drugs like acetylcholinesterase inhibitors (e.g., donepezil) boost cholinergic transmission by preventing acetylcholine breakdown, modestly improving memory and cognition in early stages. Adrenergic enhancement (e.g., via norepinephrine) affects arousal, not memory directly. Dopaminergic drugs treat Parkinson's, not Alzheimer's cognitive deficits. GABAergic drugs (e.g., benzodiazepines) inhibit cognition, worsening memory. Serotonergic agents target mood, not memory. The cholinergic deficit is a hallmark of Alzheimer's pathology, and augmenting this system remains a primary therapeutic strategy, validated by clinical outcomes.
Question 5 of 5
The patient has a potassium level of 5.9 mEq/L. The nurse is administering glucose and insulin. The patient's wife says, 'He doesn't have diabetes, why is he getting insulin?' What is the best response by the nurse?
Correct Answer: A
Rationale: Hyperkalemia (potassium 5.9 mEq/L) is treated emergently with glucose and insulin, which drive potassium into cells, temporarily lowering serum levels . Insulin facilitates this shift by enhancing cellular uptake, paired with glucose to prevent hypoglycemia—accurate and clear for the wife. Choice B misstates that potassium is excreted via blood sugar; it's redistributed, not eliminated. Choice C compares insulin to Kayexalate (which binds potassium in the gut), but safety isn't the primary rationale. Choice D incorrectly suggests renal excretion, which isn't insulin's role (diuretics or dialysis do that). Choice A explains the mechanism correctly, addressing the wife's confusion with scientific clarity.