The following are characters of venous hum EXCEPT

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Pediatric Nursing Cardiovascular Disorders Questions

Question 1 of 5

The following are characters of venous hum EXCEPT

Correct Answer: B

Rationale: In this question about venous hum in pediatric cardiovascular disorders, the correct answer is B) grades louder with mane compression. The venous hum is a benign vascular sound commonly found in healthy children. It is typically heard as a continuous murmur in the infraclavicular region. It is mostly grade l-lll or VI in the supine position and disappears or diminishes with changes in the position of the jugular vein. Option A is incorrect because a venous hum is indeed a continuous murmur heard in the infraclavicular region. Option C is also incorrect because the grading of the venous hum can vary from l-lll or even VI depending on the individual patient and their specific condition. Option D is incorrect because changes in the position of the jugular vein can influence the presence or intensity of a venous hum. Educationally, understanding the characteristics of vascular sounds like the venous hum is crucial for pediatric nurses to differentiate normal findings from abnormal ones. This knowledge helps in providing accurate assessments and appropriate care for pediatric patients with cardiovascular disorders.

Question 2 of 5

Increased pulmonary markings (increased pulmonary blood flow) is seen in

Correct Answer: B

Rationale: In pediatric nursing, understanding cardiovascular disorders is crucial for providing effective care to young patients. In this question, the correct answer is B) truncus arteriosus. Increased pulmonary markings or pulmonary blood flow is a characteristic finding in truncus arteriosus due to the single arterial trunk arising from the heart, leading to increased blood flow to the lungs. A) Pulmonary atresia involves a complete blockage of the pulmonary valve, resulting in decreased pulmonary blood flow, opposite to the presented symptom. C) Tetralogy of Fallot is characterized by decreased pulmonary blood flow due to a ventricular septal defect and overriding aorta, leading to cyanosis rather than increased pulmonary markings. D) Tricuspid atresia is associated with decreased pulmonary blood flow because of the underdeveloped right ventricle and lack of tricuspid valve function. Educationally, this question highlights the importance of recognizing key clinical manifestations of pediatric cardiovascular disorders to make accurate assessments and provide appropriate interventions. Understanding these distinctions is essential for nurses caring for pediatric patients with congenital heart conditions.

Question 3 of 5

The following are major side effects of PGE1 EXCEPT

Correct Answer: D

Rationale: Rationale: The correct answer is D) hypertension. Prostaglandin E1 (PGE1) is a medication commonly used in pediatric patients with congenital heart defects to maintain patency of the ductus arteriosus. The major side effects of PGE1 include apnea, fever, and cutaneous flushing. Apnea can occur as a result of the respiratory center being affected by PGE1. Fever is a common side effect due to the pyrogenic properties of prostaglandins. Cutaneous flushing is caused by vasodilation mediated by prostaglandins. Hypertension is not a major side effect of PGE1. In fact, PGE1 is known to cause vasodilation and can lead to hypotension rather than hypertension. It is important for pediatric nurses to be aware of the side effects of medications used in the management of cardiovascular disorders to provide safe and effective care to their patients.

Question 4 of 5

All the following are causes of chronic pulmonary venous hypertension EXCEPT

Correct Answer: C

Rationale: In pediatric nursing, understanding cardiovascular disorders, including chronic pulmonary venous hypertension, is crucial for providing effective care. In this scenario, the correct answer is option C) peripheral pulmonary stenosis. Explanation: A) Congenital mitral stenosis: This condition can lead to increased pulmonary venous pressure, contributing to pulmonary hypertension, making it a cause of chronic pulmonary venous hypertension. B) Total anomalous pulmonary venous return with obstruction: Obstruction in pulmonary venous return can elevate pulmonary venous pressure, leading to pulmonary hypertension, making it a cause of chronic pulmonary venous hypertension. C) Peripheral pulmonary stenosis: This is NOT a cause of chronic pulmonary venous hypertension. Peripheral stenosis affects the pulmonary artery, not the pulmonary veins, hence does not directly contribute to pulmonary venous hypertension. D) Cor triatriatum: This congenital heart defect can impede blood flow, causing elevated pulmonary venous pressure and contributing to pulmonary hypertension, making it a cause of chronic pulmonary venous hypertension. Educational context: Understanding the pathophysiology of different cardiovascular disorders in pediatric patients is essential for accurate diagnosis and appropriate management. Recognizing the specific causes of chronic pulmonary venous hypertension helps in developing tailored treatment plans and improving patient outcomes. This question highlights the importance of distinguishing between various cardiac anomalies and their implications on pulmonary circulation in pediatric patients.

Question 5 of 5

The MOST common cause of sudden death in competitive athletes is

Correct Answer: B

Rationale: In the context of pediatric nursing and cardiovascular disorders, understanding the most common causes of sudden death in competitive athletes is crucial for early recognition and intervention. The correct answer is B) hypertrophic cardiomyopathy. This condition is a leading cause of sudden cardiac death in young athletes due to the abnormal thickening of the heart muscle, which can disrupt the heart's electrical system and lead to fatal arrhythmias during physical activity. A) Aortic stenosis is less common in causing sudden death in athletes compared to hypertrophic cardiomyopathy. Aortic stenosis typically presents with symptoms like chest pain and shortness of breath rather than sudden death during exertion. C) Coronary artery abnormalities, while serious, are not as common a cause of sudden death in athletes as hypertrophic cardiomyopathy. These abnormalities may predispose individuals to heart attacks but are not typically associated with sudden arrhythmic death during sports. D) Myocarditis, inflammation of the heart muscle, can indeed lead to sudden death but is not as prevalent as hypertrophic cardiomyopathy among competitive athletes. Myocarditis often presents with symptoms like chest pain, fever, and fatigue before progressing to more severe complications. Educationally, recognizing the signs and symptoms of hypertrophic cardiomyopathy in young athletes is crucial for nurses working in pediatric settings. By understanding the prevalence and implications of this condition, nurses can advocate for proper cardiac screening protocols and help prevent tragic outcomes in the athletic population.

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