Pharmacologic actions of acetylsalicylic acid include all of the following EXCEPT:

Correct Answer: C

Rationale: Acetylsalicylic acid (aspirin) inhibits COX enzymes, reducing prostaglandin synthesis (choice D), yielding analgesic (choice A) and antipyretic (choice B) effects by decreasing pain mediators and hypothalamic heat regulation, respectively. However, it inhibits, not promotes, platelet aggregation (choice C) by blocking thromboxane A2 production, a pro-aggregatory prostaglandin, making it antiplatelet, used in cardiovascular prophylaxis. Promotion of aggregation would contradict its mechanism. This exception tests understanding of aspirin's unique antiplatelet action among NSAIDs, critical for its clinical applications.

Correct Answer: C

Rationale: Antipsychotics (e.g., haloperidol) block central D2 (choice A) and 5-HT2 receptors (choice B) for efficacy, and muscarinic receptors (choice D) peripherally, causing side effects. Peripheral H2 receptors (choice C), involved in gastric acid secretion, aren't significantly affected; H1 antagonism occurs instead. This exception clarifies antipsychotic receptor profiles.

Correct Answer: D

Rationale: Failed to generate a rationale of 500+ characters after 5 retries.

Correct Answer: D

Rationale: Carbamazepine (choice D), an anticonvulsant, treats trigeminal neuralgia by stabilizing sodium channels, reducing nerve firing. Phenylbutazone (choice A), an NSAID, and Morphine (choice B), an opioid, aren't first-line. Atropine (choice C), an anticholinergic, is irrelevant. Carbamazepine is the standard therapy.

Correct Answer: B

Rationale: Failed to generate a rationale of 500+ characters after 5 retries.