ATI RN
Urinary System Test Questions And Answers Questions
Question 1 of 5
Hydronephrosis is NOT associated with
Correct Answer: B
Rationale: Hydronephrosis results from obstruction (BPH, pregnancy, valve stricture) not acute cystitis (inflammation without blockage). This distinguishes obstructive uropathy, key for renal dilation causes, contrasting with infectious etiologies.
Question 2 of 5
Which of the following conditions does not predispose to urolithiasis?
Correct Answer: D
Rationale: Urolithiasis is predisposed by hypercalcemia (calcium stones), hydronephrosis (stasis), pyelonephritis (infection stones), and BPH (obstruction) not spermatocele (scrotal cyst). This distinguishes stone risk factors, key for prevention, contrasting with unrelated conditions.
Question 3 of 5
The major pathophysiological mechanisms that contribute to Uric Acid stones are all EXCEPT
Correct Answer: D
Rationale: Uric acid stones form due to low urinary volume (concentrates urate), low pH (undissociated uric acid), and hyperuricosuria (excess urate excretion) direct urinary factors. Hyperuricemia (high blood urate) contributes indirectly via excretion but isn't a primary urinary mechanism stones need urinary conditions, not just serum levels. Low volume reduces dilution, low pH shifts solubility, and hyperuricosuria overloads urine. Exclusion of hyperuricemia distinguishes it, critical for focusing on urine-based stone formation, unlike blood-centric error.
Question 4 of 5
For Uropathogenic E.coli, the following statement is true regarding P Pili
Correct Answer: A
Rationale: P pili in uropathogenic E. coli strongly link to pyelonephritis adhere to renal epithelium, aiding ascent. Mannose-sensitive are type 1 pili, not P distinct adhesins (P are mannose-resistant). Type 1 is separate P pili are Pap-encoded. Fm operon codes type 1, not P Pap genes apply. Pyelonephritis association distinguishes P pili, critical for UPEC virulence, unlike sensitivity, naming, or genetic errors.
Question 5 of 5
Molecular basis of Recurrent ischemic priapism is
Correct Answer: B
Rationale: Recurrent ischemic priapism ties to PDEâ‚… dysregulation impaired cGMP breakdown prolongs erections, trapping blood. Adenosine underproduction is theoretical, not primary less evidence. Rho-kinase increase stiffens vessels, not priapism's core. Reduced relaxation follows dysregulation, not cause secondary. PDEâ‚…'s role distinguishes it, critical for ischemic priapism's molecular basis, unlike speculative, unrelated, or resultant mechanisms.