Hormones produced by the thymus play a role in the

Correct Answer: A

Rationale: Failed to generate a rationale of 500+ characters after 5 retries.

Correct Answer: A

Rationale: Somatomedin-C (IGF-1), a liver-derived protein induced by growth hormone (GH), mediates GH's anabolic effects, promoting bone and muscle growth. Gonadotropins (FSH, LH) target gonads, not GH pathways. Prostaglandins regulate inflammation and smooth muscle, not growth. Prolactin drives lactation, not GH effects. Somatomedin-C's role as GH's effector distinguishes it, key to childhood growth and adult anabolism, unlike reproductive, inflammatory, or lactation signals.

Correct Answer: A

Rationale: Acromegaly, from pituitary GH excess post-puberty, causes bone thickening true. Addison's affects adrenals, not thyroid; hypothyroidism (low T3/T4) is true but not sole correct; Simmond's is pituitary, not adrenal. Acromegaly's pituitary origin distinguishes it, key to endocrine classification, though C is also true, A matches provided answer.

Correct Answer: C

Rationale: Rapid-acting effects typify neural responses (e.g., milliseconds), not endocrine, where hormones travel via blood (e.g., insulin), causing slow, cyclic changes (e.g., menstrual cycle) via chemical signals. Blood transport enables systemic action, slow effects reflect diffusion and receptor binding, and chemicals (hormones) drive outcomes rapid action contradicts this, distinguishing neural speed from endocrine's prolonged regulation, critical for their distinct roles.

Correct Answer: A

Rationale: Epinephrine and norepinephrine, from the adrenal medulla, drive fight-or-flight, raising heart rate, glucose, and alertness via sympathetic activation. Insulin/glucagon regulate glucose, not acute stress. Estrogen/progesterone manage reproduction, not emergency responses. Thyroxin (metabolism) and melatonin (sleep) lack rapid stress roles. Catecholamines' swift, systemic effects distinguish them, key to survival responses, unlike metabolic or reproductive hormones.