Diabetes insipidus is caused due to deficiency of hormone:

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Chapter 17 Endocrine System Test Questions Questions

Question 1 of 5

Diabetes insipidus is caused due to deficiency of hormone:

Correct Answer: B

Rationale: The correct answer is B: ADH (antidiuretic hormone). Diabetes insipidus is a condition characterized by excessive thirst and urination due to a deficiency of ADH, which regulates water balance in the body. Insulin (choice A) is related to diabetes mellitus, not diabetes insipidus. Growth hormone (choice C) and oxytocin (choice D) do not play a direct role in water balance regulation. Therefore, choice B is the most appropriate answer for this question.

Question 2 of 5

Which gland is responsible for the production of adrenaline?

Correct Answer: A

Rationale: The correct answer is A: Adrenal. The adrenal gland is responsible for producing adrenaline, also known as epinephrine, which is a hormone released in response to stress or danger. Adrenaline helps the body prepare for fight or flight responses by increasing heart rate, dilating airways, and boosting energy. The other choices, Parathyroid, Gonadal, and Pancreas, are not responsible for producing adrenaline. Parathyroid glands regulate calcium levels, gonadal glands produce sex hormones, and the pancreas produces insulin and digestive enzymes. Therefore, the adrenal gland is the correct answer for the production of adrenaline.

Question 3 of 5

A patient presents with a tumor of the adrenal gland. It is determined that the levels of renin-angiotensin is low. Which of the following would lead to increased renin-angiotensin levels?

Correct Answer: B

Rationale: The correct answer is B: Secondary hyperaldosteronism. In this scenario, the low renin-angiotensin levels indicate a feedback mechanism where the body is trying to compensate for high aldosterone levels. Secondary hyperaldosteronism, which can be caused by conditions like renal artery stenosis or heart failure, leads to increased aldosterone secretion independent of renin levels, resulting in low renin-angiotensin levels. A: Cushing's syndrome is associated with excess cortisol production, but it does not directly influence renin-angiotensin levels. C: Primary hyperaldosteronism (Conn's syndrome) is characterized by high aldosterone levels, not low renin-angiotensin levels. D: Both Cushing's syndrome and secondary hyperaldosteronism do not necessarily occur together, making this option incorrect.

Question 4 of 5

The anterior pituitary gland receives hormone messages from the hypothalamus via ________.

Correct Answer: A

Rationale: The correct answer is A: a capillary network. The hypothalamus secretes hormones into the capillary network called the hypothalamic-hypophyseal portal system. These hormones travel directly to the anterior pituitary gland to regulate the release of its own hormones. Choice B is incorrect because neurons do not directly connect the hypothalamus and pituitary gland. Choice C is incorrect because Schwann cells are involved in myelination of peripheral nerves, not hormone transport. Choice D is incorrect because although both options A and B involve communication between the hypothalamus and pituitary gland, it is specifically the capillary network that carries hormone messages in this case.

Question 5 of 5

Which one of the following is NOT typical of the changes that follow the binding of a hormone to its target cells:

Correct Answer: B

Rationale: The correct answer is B: cellular mutations occur. Hormone binding typically triggers signaling cascades that lead to cellular responses like changing plasma membrane permeability, activating/inactivating enzymes, or stimulating mitosis. Cellular mutations are not a direct result of hormone binding and are typically caused by external factors like radiation or chemicals. In this context, mutations do not happen as an immediate response to hormone binding. Therefore, option B is the correct choice. Plasma membrane permeability changes, enzyme activation/inactivation, and mitosis stimulation are all typical outcomes of hormone binding and subsequent signaling pathways.

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