Beta-blocker-induced adverse effects include all of the following EXCEPT:

Correct Answer: D

Rationale: The correct answer is D: Hyperglycemia. Beta-blockers can cause bronchoconstriction (Choice A) by blocking beta-2 receptors, leading to airway constriction. They depress myocardial contractility and excitability (Choice B) by blocking beta-1 receptors in the heart. "Supersensitivity" of beta-receptors (Choice C) can occur upon rapid withdrawal of beta-blockers due to upregulation of receptors. However, beta-blockers typically do not cause hyperglycemia (Choice D) as they may actually mask the symptoms of hypoglycemia.

Correct Answer: B

Rationale: The correct answer is B: Selegiline. Selegiline is a monoamine oxidase inhibitor used in the treatment of Parkinsonian disorders by increasing dopamine levels in the brain. Phenytoin (A) is an anticonvulsant, not used for Parkinson's. Haloperidol (C) is an antipsychotic, may worsen Parkinson's symptoms. Fluoxetine (D) is an antidepressant, not specific for Parkinson's treatment. Therefore, Selegiline is the most appropriate choice due to its mechanism of action in increasing dopamine levels to alleviate Parkinsonian symptoms.

Correct Answer: B

Rationale: The correct answer is B: Dilatation of the biliary duct. Morphine does not cause dilation of the biliary duct; instead, it can lead to biliary spasm and constriction. Morphine commonly causes constipation by decreasing gut motility, urinary retention by affecting the bladder sphincter, and bronchiolar constriction by acting on the respiratory system. These effects are well-documented pharmacological actions of morphine.

Correct Answer: A

Rationale: The correct answer is A: Carbamazepine. Carbamazepine has a dual mechanism of action, functioning as both an antiseizure medication and an analgesic. It is commonly used to treat neuropathic pain and trigeminal neuralgia. Carbamazepine's analgesic properties stem from its ability to block voltage-gated sodium channels, reducing neuronal excitability and pain transmission. Summary of other choices: B: Ethosuximide - Primarily used for absence seizures, lacks analgesic properties. C: Phenytoin - Effective against various seizure types but does not have significant analgesic effects. D: Clonazepam - A benzodiazepine used as an antiseizure medication, but does not possess analgesic properties.

Correct Answer: A

Rationale: Step-by-step rationale: 1. Monoamine Oxidase A (MAO-A) is an enzyme responsible for metabolizing neurotransmitters such as norepinephrine, serotonin, and tyramine. 2. MAO-A specifically targets norepinephrine and serotonin, making choice A correct. 3. Choices B, C, and D are incorrect as they incorrectly describe the functions and selectivity of MAO-A. In summary, MAO-A is responsible for norepinephrine, serotonin, and tyramine metabolism, making choice A the correct answer.