Atrial natriuretic hormone (ANH) is produced by cells of the

Correct Answer: B

Rationale: ANH, from right atrial cells, responds to stretch (high volume), lowering pressure via natriuresis cardiac origin. Kidney tubules respond, not produce. Adrenal cortex makes aldosterone, opposing ANH. Pancreas focuses on glucose. Atrial production distinguishes ANH, key to heart-volume regulation, unlike renal, adrenal, or metabolic sources.

Correct Answer: B

Rationale: Synaptic signaling occurs when neurons release neurotransmitters (e.g., acetylcholine) across synapses to influence adjacent neurons fast, local neural communication. Autocrine involves cells signaling themselves (e.g., growth factors). Paracrine affects nearby cells (e.g., histamine), not specifically neurons. Endocrine uses blood for distant targets (e.g., insulin), not neuron-neighbor. Synaptic specificity distinguishes it, key to nervous system function, unlike self, nearby, or systemic signaling.

Correct Answer: B

Rationale: Adrenal medulla secretes adrenaline and noradrenaline (epinephrine/norepinephrine) catecholamines for fight-or-flight. T4/T3 (thyroid) regulate metabolism, not medullary. Insulin/glucagon (pancreas) manage glucose, not adrenal. Aldosterone/cortisol (adrenal cortex) are steroids, not medulla. Adrenaline-noradrenaline's neural-endocrine role distinguishes them, critical for acute stress, unlike thyroid, pancreatic, or cortical hormones.

Correct Answer: B

Rationale: The hypothalamus secretes releasing hormones like PRH (prolactin), CRH (corticotropin), and TRH (thyrotropin) not FSH, which the anterior pituitary produces under hypothalamic GnRH control. This distinction clarifies hypothalamic regulatory roles, vital for NEET's pituitary-hypothalamic axis understanding, contrasting with pituitary effectors.