ADH will stimulate water reabsorption by the kidneys and the blood vessels; thus blood pressure.

Correct Answer: C

Rationale: ADH increases kidney water reabsorption and constricts blood vessels (V1 receptors), raising blood pressure volume and resistance boost. Decreased reabsorption contradicts ADH diuresis lowers pressure. Dilation would drop pressure, opposing constriction. Increased reabsorption with constriction distinguishes ADH's action, critical for pressure/fluid regulation, unlike decreasing or dilatory errors.

Correct Answer: B

Rationale: Calcitonin, from thyroid C-cells, lowers high blood calcium by depositing it into bones opposes resorption. PTH (parathyroid) raises calcium, opposite effect. Oxytocin (hypothalamus) aids labor, unrelated. Glucagon (pancreas) raises glucose, not calcium. Calcitonin's bone storage distinguishes it, critical for calcium balance, unlike raising, reproductive, or glucose hormones.

Correct Answer: D

Rationale: Aldosterone increases blood pressure by targeting kidney tubules for sodium/water reabsorption (less urine) and potassium excretion mineralocorticoid action. Decreasing blood pressure contradicts raises it via volume. Mineralocorticoid status and urine reduction are true. Pressure increase distinguishes aldosterone, critical for RAAS, unlike erroneous decrease.

Correct Answer: D

Rationale: Glucagon (pancreas) raises blood glucose via glycogenolysis and promotes fat/protein use (sparing glucose), but doesn't store glucose as glycogen insulin does that (glycogenesis). Storage contradicts glucagon's role mobilizes energy. Pancreatic origin, glucose increase, and sparing are true. Non-storage distinguishes glucagon, key to its catabolic action, unlike anabolic error.

Correct Answer: B

Rationale: Aldosterone (adrenal) regulates sodium/potassium, not fuel (glucose/fat) electrolyte focus. Insulin lowers glucose, glucagon raises it, cortisol mobilizes fuel (gluconeogenesis, lipolysis) all metabolic. Aldosterone's non-metabolic role distinguishes it, key to fluid balance, unlike glucose/fat-regulating hormones.