ATI RN
Pharmacology Across the Lifespan ATI Quizlet Questions
Question 1 of 5
A 48-year-old male presents to the emergency department with chest pain for the past 2 days. The pain is nonradiating and burning in nature. An ECG is normal, and serial troponins are negative. The physician makes the diagnosis of GERD. What is the most potent therapy for the prevention of GERD?
Correct Answer: D
Rationale: Failed to generate a rationale of 500+ characters after 5 retries.
Question 2 of 5
A 58-year-old man with a history of occasional GERD presents to the clinic with diarrhea for the past 3 months. He has not been sick and feeling quite well otherwise. Further history uncovers that 3 months ago, he switched the type of antacid he uses for reflux. He stopped the antacid and calls back 1 month later, saying he has not had diarrhea since then. Which of the following antacids was most likely causing the patient’s diarrhea?
Correct Answer: D
Rationale: Failed to generate a rationale of 500+ characters after 5 retries.
Question 3 of 5
A 54-year-old woman with a history of migraine headaches has begun on therapy with Topamax as prophylaxis. She is poorly communicative and a poor historian. Which of the following would be important coexisting conditions to know about?
Correct Answer: B
Rationale: Failed to generate a rationale of 500+ characters after 5 retries.
Question 4 of 5
The potency of inhaled anesthetics is defined as the minimum alveolar concentration. Five anesthetic gases are infused into 2-kg rats denoted as gas A through gas E with the MACs noted. Which of the following gases would likely be nitrous oxide?
Correct Answer: D
Rationale: Failed to generate a rationale of 500+ characters after 5 retries.
Question 5 of 5
A 35-year-old woman is diagnosed with gastroesophageal reflux disease. Omeprazole is administereWhich of the following is the most likely mechanism of action of this drug?
Correct Answer: D
Rationale: Omeprazole, a proton pump inhibitor (PPI), treats GERD by irreversibly inhibiting H⁺-K⁺ ATPase in parietal cells, blocking acid secretion into the stomach lumen. Gastrin receptor blockade isn’t its mechanism (gastrin stimulates acid indirectly). H₁ receptors relate to allergies, M₂ receptors to parasympathetic effects, and gastrin synthesis inhibition (E) isn’t a PPI action—none reduce acid as directly or potently as PPIs.