ATI RN
ATI Pharmacology Book Questions
Question 1 of 5
A 21-year-old male college student presents to the emergency department with hives over his chest and arms. The history and physical exam reveals that he used a new laundry detergent and he was having an allergic reaction. The physician wants to prescribe an antihistamine that will not sedate the patient because he has to study for a test. What is the most appropriate treatment?
Correct Answer: D
Rationale: Allergic hives from detergent need a non-sedating antihistamine. Fexofenadine -blocks H1 receptors without drowsiness, ideal for studying. Chlorpheniramine , dimenhydrinate , and diphenhydramine sedate. Ipratropium (E) is irrelevant. Fexofenadine ensures efficacy and alertness.
Question 2 of 5
A client is prescribed methotrexate (Rheumatrex) for rheumatoid arthritis. Which teaching point should the nurse emphasize?
Correct Answer: A
Rationale: Methotrexate, a DMARD, treats RA but risks renal toxicity from crystal formation. Drinking plenty of water ensures excretion, reducing this risk, a critical teaching point. Milk doesn't help'absorption is unaffected. Green leafy vegetables increase folate, which methotrexate depletes, but supplementation is controlled. Stopping if better risks flare-ups. Hydration aligns with methotrexate's pharmacokinetics, vital in RA where long-term safety matters, making A the priority to emphasize for renal protection.
Question 3 of 5
When taking Digoxin, low levels of what can cause
Correct Answer: A
Rationale: When taking digoxin, low levels of potassium can cause an increased risk of digoxin toxicity. This is because digoxin and potassium compete for binding sites on the sodium-potassium pump in cardiac cells. Low potassium levels can lead to enhanced binding of digoxin to these pumps, resulting in increased concentrations of digoxin within the cells and potential toxicity. Therefore, it is important to monitor potassium levels regularly while taking digoxin to prevent complications.
Question 4 of 5
The nurse teaches the client about the difference between oral and nasal decongestants. The nurse evaluates that learning has been effective when the client makes which statement?
Correct Answer: A
Rationale: Oral decongestants (e.g., pseudoephedrine) act systemically, raising blood pressure via vasoconstriction, a key difference from nasal decongestants (e.g., oxymetazoline), which act locally but risk rebound congestion. The statement 'Oral decongestants can cause hypertension' shows the client grasps this systemic effect, indicating effective teaching. Nasal decongestants aren't safe for a month ; they cause rebound after 3-5 days. Only nasal decongestants typically cause rebound , not oral. Oral efficacy varies, not inherently superior. Choice A reflects accurate understanding of oral decongestants' broader impact, critical for safe use, making it the best learning indicator.
Question 5 of 5
A 30-year-old male patient who is treated with haloperidol for his diagnosis of schizophrenia is considered to be well-managed symptomatically for his psychotic symptoms. However, he is reporting restlessness, the inability to sit still at the dinner table, and his family notices that he is pacing up and down the hallway frequently. Of the following, which is the best medication to treat this antipsychotic-induced akathisia?
Correct Answer: E
Rationale: Haloperidol's D2 blockade causes akathisia—restlessness and pacing—as an EPS. Propranolol, a beta-blocker, reduces akathisia by modulating catecholamine activity, a first-line treatment per guidelines, unlike anticholinergics (benztropine) which target dystonia/parkinsonism. Dantrolene treats muscle spasticity, not akathisia. Amoxapine, an antidepressant, worsens psychosis. Bromocriptine, a dopamine agonist, risks psychosis exacerbation. Propranolol's efficacy, safety, and specificity for akathisia make it the best choice, replacing D as instructed.