ATI RN
Pharmacology Across the Lifespan Questions
Question 1 of 5
A 19-year-old male presents to the emergency room with a broken ankle after a fall. He is given hydrocodone for the pain and, soon after, his stomach becomes upset. He has vomited once. The patient is given ondansetron to treat his nausea. What is the mechanism of action of ondansetron?
Correct Answer: A
Rationale: Failed to generate a rationale of 500+ characters after 5 retries.
Question 2 of 5
A 62-year-old man presents to his primary care physician for follow-up. He has a history of an atrial arrhythmia. He takes multiple medications but does not know the names of them. He now complains of headache, dizziness, and tinnitus. Which one of the following antiarrhythmic drugs is the most likely cause?
Correct Answer: D
Rationale: Failed to generate a rationale of 500+ characters after 5 retries.
Question 3 of 5
A 39-year-old woman with chronic migraine headaches manages her condition with ibuprofen 200 mg. Most days, she does well with 200 to 400 mg. Adverse reactions to this therapy include which of the following?
Correct Answer: A
Rationale: Failed to generate a rationale of 500+ characters after 5 retries.
Question 4 of 5
A 33-year-old woman patient needs to be anesthetized before a dilation and curettage for abnormal uterine bleeding. Her anesthesia is induced initially with an inhaled, nonflammable anesthetic that contains no halogenated carbons. Of the following, which agent best fits this description?
Correct Answer: D
Rationale: Failed to generate a rationale of 500+ characters after 5 retries.
Question 5 of 5
A 20-year-old woman comes to the emergency department after ingesting at least 30 tablets of an unknown drug. Initial physical examination shows no abnormalities. Thirty-six hours later, serum AST activity is 1500 U/L, and serum ALT activity is 2000 U/L. The drug this patient ingested is most likely which of the following?
Correct Answer: A
Rationale: Elevated AST and ALT (1500 and 2000 U/L) 36 hours post-ingestion indicate acute liver injury, characteristic of acetaminophen overdose. Acetaminophen is metabolized to NAPQI, which depletes glutathione and causes centrilobular hepatic necrosis if untreateAspirin causes salicylism (tinnitus, acidosis), chlorpheniramine sedation, ibuprofen GI upset, and prednisone (E) adrenal suppression—none primarily cause delayed hepatotoxicity like this.