What should be monitored when a patient is taking NSAIDs? (Select one that does not apply)

Correct Answer: A

Rationale: The correct answer is A (Gds) because monitoring gastrointestinal (GI) adverse effects is essential when a patient is taking NSAIDs due to the increased risk of ulcers, bleeding, and perforation. B: CNS adverse effects are potential side effects of NSAIDs, so monitoring for symptoms like headache, dizziness, and confusion is necessary. C: Cardiac adverse effects such as increased risk of heart attack and stroke should be monitored in patients taking NSAIDs. D: Renal adverse effects like decreased kidney function and fluid retention are common with NSAIDs and warrant monitoring. In summary, choice A (Gds) does not apply because GI adverse effects are a crucial consideration when monitoring patients on NSAIDs, making it the correct answer.

Correct Answer: A

Rationale: The correct answer is A: Cyclophosphamide. Cyclophosphamide can cause hemorrhagic cystitis as a side effect, which is less likely to occur if taken in the morning due to the diurnal variation in bladder toxicity. Methotrexate, Leflunomide, and Adalimumab are not associated with hemorrhagic cystitis and do not require specific timing for administration to prevent this side effect.

Correct Answer: A

Rationale: Rationale for Correct Answer (A): Antihistamines do not help poison ivy. Poison ivy rash is a type IV delayed hypersensitivity reaction, not a histamine-mediated reaction. Therefore, antihistamines will not provide relief. The most appropriate response is to educate the client on the ineffectiveness of antihistamines for poison ivy. Summary of Incorrect Choices: B: There are different antihistamines to try. This is incorrect because antihistamines are not effective for poison ivy rash. C: You should be seen in the clinic right away. Immediate clinic visit is not necessary unless there are severe symptoms or complications. D: You will need to take some IV steroids. IV steroids are not the first-line treatment for poison ivy rash. Topical steroids are usually sufficient.

Correct Answer: D

Rationale: The correct answer is D: Hemachromatosis. In hemachromatosis, the body absorbs and stores too much iron, leading to high transferrin iron saturation. Rationale: 1. In hemachromatosis, the body absorbs excess iron from the diet, causing high iron levels in the blood. 2. Transferrin is a protein that transports iron in the blood. In hemachromatosis, the high iron levels saturate the transferrin, leading to high percent transferrin iron saturation. 3. Anemia of chronic disease (A) is characterized by low iron levels and low transferrin iron saturation. 4. Iron deficiency (B) results in low iron levels and low transferrin iron saturation. 5. Pernicious anemia (C) is caused by vitamin B12 deficiency, not iron overload, leading to normal to low transferrin iron saturation. Summary: Hemachromatosis leads to high

Correct Answer: C

Rationale: The correct answer is C: Artificially acquired active immunity. The tetanus toxoid vaccine contains inactivated tetanus toxin, which stimulates the immune system to produce antibodies against tetanus. This process results in the person developing immunity to tetanus in the future. It is considered active immunity because the individual's immune system is actively involved in producing antibodies. A: Artificially acquired passive immunity involves receiving pre-formed antibodies, which provide immediate but temporary protection. This is not the case with the tetanus toxoid vaccine. B: Naturally acquired active immunity occurs when a person is exposed to the pathogen and develops immunity through their own immune response. This is not the case with the tetanus toxoid vaccine. D: Naturally acquired passive immunity involves receiving antibodies from another source, such as through breastfeeding. This is not how the tetanus toxoid vaccine works.