ATI LPN
Renal System Questions Quizlet Questions
Question 1 of 5
Diagnostic testing of an adult patient reveals renal glycosuria. The nurse should recognize the need for the patient to be assessed for what health problem?
Correct Answer: C
Rationale: Renal glycosuria often indicates diabetes mellitus (C), where high blood glucose exceeds reabsorption capacity. It's benign alone but not linked to DI (A), SIADH (B), or carcinoma (D), making C the assessment focus.
Question 2 of 5
The nurse is assessing a patient suspected of having developed acute glomerulonephritis. The nurse should expect to address what clinical manifestation that is characteristic of this health problem?
Correct Answer: A
Rationale: The primary presenting feature of acute glomerulonephritis is hematuria (blood in the urine), which may be microscopic (identifiable through microscopic examination) or macroscopic or gross (visible to the eye). Proteinuria, primarily albumin, which is present, is due to increased permeability of the glomerular membrane. Blood urea nitrogen (BUN) and serum creatinine levels may rise as urine output drops. Some degree of edema and hypertension is noted in most patients.
Question 3 of 5
A patient on the medical unit has a documented history of polycystic kidney disease (PKD). What principle should guide the nurses care of this patient?
Correct Answer: B
Rationale: PKD is incurable and care focuses on support and symptom control. It is not self-limiting and is not treated surgically or with lithotripsy.
Question 4 of 5
The nurse is collecting data for a patient who returned from dialysis. What finding is anticipated?
Correct Answer: B
Rationale: Weight loss occurs due to fluid removal during dialysis.
Question 5 of 5
Which of the following promotes sodium retention and potassium loss from the blood across the wall of the distal convoluted tubule?
Correct Answer: C
Rationale: Aldosterone (C) increases sodium reabsorption and potassium secretion in the distal tubule, regulating electrolytes. Renin (A) triggers the RAAS, angiotensin II (B) stimulates aldosterone, and angiotensin I (D) is a precursor, making C the direct effector.