A client with acute asthma is prescribed short-term corticosteroid therapy. What is the rationale for the use of steroids in clients with asthma?

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Question 1 of 5

A client with acute asthma is prescribed short-term corticosteroid therapy. What is the rationale for the use of steroids in clients with asthma?

Correct Answer: C

Rationale: Corticosteroids (C) in acute asthma reduce inflammation, decreasing bronchial edema and mucus that obstruct airflow an anti-inflammatory effect critical for reversing exacerbation severity. They don't bronchodilate (A) beta-agonists do that or act as expectorants (B) to clear mucus; they lessen its production. Infection prevention (D) isn't their role; they may increase risk long-term. Short-term use (e.g., prednisone) complements bronchodilators, targeting inflammation's root, not just symptoms, key in nursing rationale to explain their delayed but essential action in restoring airway patency and preventing relapse.

Question 2 of 5

A patient came to the clinic with asthmatic attack, his body did not respond to the drugs and it is found that he had hypoxemia. What do we call this condition?

Correct Answer: D

Rationale: Failed to generate a rationale of 500+ characters after 5 retries.

Question 3 of 5

15 female patient present to the physician with breathlessness cough and sputum production the patient diagnosed with bronchial asthma which of the following would be found if we do sputum analysis?

Correct Answer: A

Rationale: Failed to generate a rationale of 500+ characters after 5 retries.

Question 4 of 5

The lungs are attached to the walls of the thorax by means of the:

Correct Answer: B

Rationale: The parietal pleural membrane (B) attaches lungs to the thorax . This serous layer lines the chest wall, adhering via negative pressure (-5 mmHg) to the visceral pleura (C), encasing lungs. The pericardium (A) surrounds the heart, not lungs. Mesentery (D) supports abdominal organs. B's pleural linkage unlike A's cardiac or D's digestive role maintains lung position during breathing's 500 mL tidal shifts, per physiology (Page 1).

Question 5 of 5

Regarding pulmonary vascular resistance, which one of the following is true?

Correct Answer: B

Rationale: Pulmonary vascular resistance (PVR) dips at low lung volumes (B) near FRC (~2.5 L), where capillaries stretch optimally . At high volumes (A), extra-alveolar vessels compress, raising PVR; at very low volumes, collapse hikes it too FRC's the sweet spot (~0.1 mmHg/L/min). Increased PVR (C) strains the right heart (e.g., fibrosis, Q10), but it's true, not the query's focus. PVR isn't routine in spirometry (D, false). B's low-volume minimum reflects physiology's balance unlike A's high-volume rise or D's testing mismatch per vascular dynamics.

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