ATI LPN
Pharmacology and the Nursing Process 10th Edition Test Bank
Chapter 31 : Thyroid and Antithyroid Drugs Questions
Question 1 of 5
A patient, newly diagnosed with hypothyroidism, receives a prescription for a thyroid hormone replacement drug. The nurse assesses for which potential contraindication to this drug?
Correct Answer: D
Rationale: Thyroid hormone replacement is contraindicated in recent myocardial infarction due to increased cardiac workload, which could exacerbate heart conditions. Infection, diabetes, and liver disease are not absolute contraindications.
Question 2 of 5
A patient with hypothyroidism is given a prescription for levothyroxine. When the nurse explains that this is a synthetic form of the thyroid hormone, he states that he prefers to receive more 'natural' forms of drugs. What will the nurse explain to him about the advantages of levothyroxine?
Correct Answer: D
Rationale: Levothyroxine's long half-life allows for convenient once-daily dosing, improving adherence compared to natural forms. It does not necessarily have stronger effects, lower cost, or fewer GI adverse effects.
Question 3 of 5
The order reads, 'Give levothyroxine, 200 mg, PO once every morning.' Which action by the nurse is correct?
Correct Answer: D
Rationale: Levothyroxine is dosed in micrograms (mcg), not milligrams (mg). A 200 mg dose would be a thousandfold overdose, so the nurse must question the order to ensure it is corrected to 200 mcg.
Question 4 of 5
During a teaching session for a patient on antithyroid drugs, the nurse will discuss which dietary instructions?
Correct Answer: B
Rationale: Antithyroid drugs work best when iodine intake is minimized, as iodine-rich foods can reduce drug effectiveness. Iodized salt, fluid restriction, and increased sodium/potassium intake are not recommended.
Question 5 of 5
A patient who is taking propylthiouracil for hyperthyroidism wants to know how this medicine works. Which explanation by the nurse is accurate?
Correct Answer: B
Rationale: Propylthiouracil inhibits thyroid hormone synthesis by blocking iodine incorporation, slowing hormone formation. It does not promote hormone formation, destroy cells, or inactivate existing hormone.